Masthead

nCounter®
Glial Profiling Panel

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Helping Your Research

Comprehensively study the role of astrocytes, microglia, and oligodendrocytes in health and disease with the nCounter Glial Profiling Panel. Decipher the complex interplay between glial cells, peripheral immune cells, and neurons that underlies neurodegenerative & neuroinflammatory disorders and neurotrauma such as stroke, spinal cord injury, and traumatic brain injury.

Counter Glial Profiling

How It Works

The Glial Profiling panel covers comprehensive glial cell biology involved in both homeostasis and disease and can be run on its own or paired with the Neuropathology or Neuroinflammation panels.

01:

Profile 770 human or mouse genes across 50+ pathways involved in glial cell biology:

  • Cell stress & Damage Response
  • Pathways Regulating Glia
  • Inflammation & Peripheral Immune Invasion
  • Glial Cell Homeostasis & Activation
  • Neurotransmission
02:

Quantify the relative abundance of 5 CNS cell types and 14 peripheral immune cells

03:

Customizable with Panel Plus option – add up to 55 genes of your choice

Panel Selection Tool

Find the gene expression panel for your research with easy to use panel pro

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Product Information

Functional Annotations
Cell Type Gene Coverage
Product Specifications
Catalog Information
Functional Annotations
Cell Type Gene Coverage
Product Specifications
Catalog Information

Related Resources

See All Resources
Product Bulletin Glial Panel – Product Bulletin
Flyer Glial Cell Publications – Flyer
Blog Rediscovering Microglia: Dr. Oleg Butovsky Discusses the Untapped Potential of Microglia in Neurodegenerative Diseases

Publications

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Co-activation of Sonic hedgehog and Wnt signaling in murine retinal precursor cells drives ocular lesions with features of intraocular medulloepithelioma.

Intraocular medulloepithelioma (IO-MEPL) is a rare embryonal ocular neoplasm, prevalently occurring in children. IO-MEPLs share histomorphological features with CNS embryonal tumors with multilayered rosettes (ETMRs), referred to as intracranial medulloepitheliomas.

Characterization of the therapeutic effect of antibodies targeting the Ebola glycoprotein using a novel BSL2-compliant rVSVΔG-EBOV-GP infection model.

Ebola virus (EBOV) infections cause haemorrhagic fever, multi-organ failure and death, and survivors can experience neurological sequelae. Licensing of monoclonal antibodies targeting EBOV glycoprotein (EBOV-GP) improved its prognosis, however, this treatment is primarily effective during early stages of disease and its effectiveness in reducing neurological sequela remains unknown.

MMP9 Differentially Regulates Proteins Involved in Actin Polymerization and Cell Migration during TGF-β-Induced EMT in the Lens.

Fibrotic cataracts have been attributed to transforming growth factor-beta (TGF-β)-induced epithelial-to-mesenchymal transition (EMT). Using mouse knockout (KO) models, our laboratory has identified MMP9 as a crucial protein in the TGF-β-induced EMT process.

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